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Microglia roles in synaptic plasticity and myelination in homeostatic conditions and neurodevelopmental disorders
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The NF–κB pathway is initiated by the degradation of IκBα, enabling NF-κB to translocate to the nucleus and promote the transcription of pro-inflammatory cytokines such as TNF-α, IL-1β, and IL-6, which further damage endothelial cells and disrupt tight junction proteins (Figure 3) [188,189,190]
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